Baroreceptors (or archaically, pressoreceptors) are sensors located in the carotid sinus (at the bifurcation of external and internal carotids) and in the aortic arch. They sense the blood pressure and relay the information to the brain, so that a proper blood pressure can be maintained.
Baroreceptors are a type of mechanoreceptor sensory neuron that are excited by a stretch of the blood vessel. Thus, increases in the pressure of blood vessel triggers increased action potential generation rates and provides information to the central nervous system. This sensory information is used primarily in autonomic reflexes that in turn influence the heart cardiac output and vascular smooth muscle to influence vascular resistance.[1] Baroreceptors act immediately as part of a negative feedback system called the
The arterial baroreflex buffers acute fluctuations in blood pressure that occur during posture, stress, or other maneuvers. When blood pressure rises, vascular distension is transduced into nervous electrical activity, triggering reflex parasympathetic activation and sympathetic inhibition.
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Baroreceptors (or archaically, pressoreceptors) are sensors located in the carotid sinus (at the bifurcation of external and internal carotids) and in the aortic arch. They sense the blood pressure and relay the information to the brain, so that a proper blood pressure can be maintained.
Baroreceptors are a type of mechanoreceptor sensory neuron that are excited by a stretch of the blood vessel. Thus, increases in the pressure of blood vessel triggers increased action potential generation rates and provides information to the central nervous system. This sensory information is used primarily in autonomic reflexes that in turn influence the heart cardiac output and vascular smooth muscle to influence vascular resistance.[1] Baroreceptors act immediately as part of a negative feedback system called the
The arterial baroreflex buffers acute fluctuations in blood pressure that occur during posture, stress, or other maneuvers. When blood pressure rises, vascular distension is transduced into nervous electrical activity, triggering reflex parasympathetic activation and sympathetic inhibition.